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Minocycline reduces reperfusion injury by inhibiting matrix metalloproteinases mmps and microglia activity after cerebral ischemia.
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Minocycline was not mutagenic in tu qi 2010 vitro in a bacterial reverse mutation assay ames test or cho hgprt mammalian cell assay in the presence or absence of metabolic activation.
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Skipping doses or not taking all doses of minocycline hydrochloride extended-release tablets may make the treatment not work as well.
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